The genetic control of liver cAMP levels in mice

William Lafuse; Daniel Meruelo; Michael Edidin

doi:10.1007/BF01570393

The genetic control of liver cAMP levels in mice

William Lafuse, Daniel Meruelo, Michael Edidin

Krieger School of Arts and Sciences

Research output: Contribution to journal › Article › peer-review

12 Scopus citations

Abstract

Steady-state level of liver 3′,5′-cyclic monophosphate, cAMP, has been shown to be under genetic control linked to the mouse H-2 complex. Liver cAMP levels are associated with H-2 haplotype in fully segregating crosses of strains C3H and C57BL/10. In crosses involving strain A, other loci have an effect that swamps that of H-2. Results with H-2 recombinants indicate that liver cAMP levels are affected by more than one H-2-linked locus.

Original language	English (US)
Pages (from-to)	57-65
Number of pages	9
Journal	Immunogenetics
Volume	9
Issue number	1
DOIs	https://doi.org/10.1007/BF01570393
State	Published - Dec 1 1979

ASJC Scopus subject areas

Immunology
Genetics

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10.1007/BF01570393

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AU - Edidin, Michael

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AB - Steady-state level of liver 3′,5′-cyclic monophosphate, cAMP, has been shown to be under genetic control linked to the mouse H-2 complex. Liver cAMP levels are associated with H-2 haplotype in fully segregating crosses of strains C3H and C57BL/10. In crosses involving strain A, other loci have an effect that swamps that of H-2. Results with H-2 recombinants indicate that liver cAMP levels are affected by more than one H-2-linked locus.

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The genetic control of liver cAMP levels in mice

Abstract

ASJC Scopus subject areas

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