The apoptosis inhibitor ARC alleviates the ER stress response to promote β-cell survival

Keyphrases

• Cell Survival 100%
• Inhibitor of Apoptosis Proteins 100%
• Endoplasmic Reticulum Stress Response 100%
• Endoplasmic Reticulum 66%
• Palmitate 66%
• C/EBP Homologous Protein 66%
• Cell Failure 66%
• Loss Function 33%
• Genetic Loss 33%
• Mitochondria 33%
• Protective Effect 33%
• Cell Death 33%
• Thapsigargin 33%
• Apoptosis 33%
• Death Receptor 33%
• Insulin Resistance 33%
• Induced Apoptosis 33%
• Cell Loss 33%
• Cell Apoptosis 33%
• Inducer 33%
• Type 2 Diabetes Mellitus (T2DM) 33%
• Inositol 33%
• Cell Death Pathways 33%
• Human Cells 33%
• Insulin Secretion 33%
• Tunicamycin 33%
• Physiological Concentrations 33%
• Gain Loss 33%
• Apoptosis Repressor with Caspase Recruitment Domain 33%
• Isolated Islets 33%
• Endocrine Pancreas 33%
• Function Approach 33%
• Stress Response Pathway 33%
• Skeletal Myocytes 33%
• Genetic Gain 33%
• Protein Induction 33%
• Unusual Properties 33%
• Protein Kinase R-like Endoplasmic Reticulum Kinase 33%

Biochemistry, Genetics and Molecular Biology

• Cell Survival 100%
• Endoplasmic Reticulum Stress 100%
• Cell Death 66%
• Inhibitor of Apoptosis 66%
• Protein Kinases 33%
• RNA 33%
• Mouse 33%
• Mitochondrion 33%
• Eicosanoid Receptor 33%
• Insulin Resistance 33%
• Synapsin I 33%
• Kinase 33%
• Phosphotransferase 33%
• Insulin Release 33%
• Tunicamycin 33%
• Inositol 33%
• Repressor 33%
• Cell Loss 33%
• CARD Domain 33%
• Genetic Gain 33%
• Protein Induction 33%