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Signal integration of IFN-I and IFN-II with TLR4 involves sequential recruitment of STAT1-Complexes and NFκB to enhance pro-inflammatory transcription
Anna Piaszyk-Borychowska
, Lajos Széles
, Attila Csermely
, Hsin Chien Chiang
, Joanna Wesoły
, Chien Kuo Lee
,
Laszlo Nagy
, Hans A.R. Bluyssen
School of Medicine
Research output
:
Contribution to journal
›
Article
›
peer-review
9
Scopus citations
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Dive into the research topics of 'Signal integration of IFN-I and IFN-II with TLR4 involves sequential recruitment of STAT1-Complexes and NFκB to enhance pro-inflammatory transcription'. Together they form a unique fingerprint.
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Immunology and Microbiology
interferon
100%
TLR4
100%
STAT1
100%
Complement Factor B
100%
DNA Responsive Element
13%
Atherosclerosis
13%
Immunocompetent Cell
8%
Dendritic Cell
8%
Vascular Smooth Muscle Cell
8%
Interferon Stimulated Gene Factor 3
8%
Gene Expression
4%
Mouse
4%
Macrophage
4%
Transcription Factors
4%
Transcription Initiation
4%
Promoter Region
4%
Gene Expression Regulation
4%
Histone Acetylation
4%
Blood Vessels
4%
Genetic Transcription
4%
Inflammatory Disorder
4%
Vasculitis
4%
Interferon Regulatory Factor 9
4%
Keyphrases
Interferon-I
100%
IRF9
6%