Possible mechanisms for horizontal gaze deviation and lateropulsion in the lateral medullary syndrome

David Solomon, Steven L. Gaietta, Grant T. Liu

Research output: Contribution to journalArticlepeer-review

27 Scopus citations

Abstract

We report a patient who developed conjugate horizontal gaze deviation and ipsipulsion of saccades from a lateral medullary infarction. Recent evidence suggests that the gaze deviation may result from increased inhibition of the ipsilateral vestibular nucleus and ipsipulsion of sac-cades from decreased excitation of the contralateral ocular premotor areas of the brainstem reticular formation. Interruption of the olivocerebellar pathways may account for both of these ocular motor abnormalities.

Original languageEnglish (US)
Pages (from-to)26-30
Number of pages5
JournalJournal of Neuro-Ophthalmology
Volume15
Issue number1
DOIs
StatePublished - Mar 1995
Externally publishedYes

Keywords

  • Gaze deviation
  • Lateral medullary (Wallenberg) syndrome
  • Lateropulsion

ASJC Scopus subject areas

  • Ophthalmology
  • Clinical Neurology

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