Abstract
A population of neurons in patients with familial amyotrophic lateral sclerosis have multiple abnormalities, including protein aggregation and mitochondrial dysfunction. Two studies now reveal interactions between these two defects.
Original language | English (US) |
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Pages (from-to) | 905-906 |
Number of pages | 2 |
Journal | Nature medicine |
Volume | 10 |
Issue number | 9 |
DOIs | |
State | Published - Sep 2004 |
ASJC Scopus subject areas
- General Biochemistry, Genetics and Molecular Biology