Effect of iron and ascorbate on cyclosporine-induced oxidative damage of kidney mitochondria and microsomes

Keyphrases

• Cyclosporine 100%
• Oxidative Damage 100%
• Microsomes 100%
• Kidney Mitochondria 100%
• Effect of Iron 100%
• Fe2+ 33%
• Mitochondria 16%
• Lipids 16%
• Kidney Epithelial Cells 16%
• Absorbance Spectra 8%
• Hydrogen Peroxide 8%
• Epithelial Cells 8%
• Stimulatory Effect 8%
• Kidney Cortex 8%
• Nephrotoxicity 8%
• Hydrogen Peroxide Production 8%
• Malondialdehyde 8%
• Glutathione Peroxidase (GSH-Px) 8%
• Manganese Superoxide Dismutase (MnSOD) 8%
• Tissue Components 8%
• Wavelength Range 8%
• 2-deoxy-D-ribose (2dDR) 8%
• Peroxidative 8%
• Protein Thiols 8%
• Viability Loss 8%
• Deoxyribose Degradation 8%
• Cell Viability Loss 8%
• Mitochondrial Thiols 8%
• Damage Effect 8%

Biochemistry, Genetics and Molecular Biology

• Mitochondrion 100%
• Ciclosporin 100%
• Ascorbic Acid 100%
• Vitamin C 100%
• Hydrogen Peroxide 16%
• Lipid 16%
• Cell Viability 8%
• Glutathione Peroxidase 8%
• Deoxyribose 8%
• Thiol 8%

Pharmacology, Toxicology and Pharmaceutical Science

• Ciclosporin 100%
• Ascorbic Acid 100%
• Ferrous Ion 33%
• Hydrogen Peroxide 16%
• Ethylenediaminetetraacetic Acid 16%
• Pentetic Acid 16%
• Lipid 16%
• Cell Viability 8%
• Malonaldehyde 8%
• Nephrotoxicity 8%
• Dimethyl Sulfoxide 8%
• Glutathione Peroxidase 8%
• 2 Deoxyribose 8%
• Ferrous Ascorbate 8%
• Thiol 8%