Distinct Mechanisms of Tumor Resistance to NK Killing: Of Mice and Men

Drew M. Pardoll

doi:10.1016/j.immuni.2015.04.007

Distinct Mechanisms of Tumor Resistance to NK Killing: Of Mice and Men

Drew M. Pardoll

School of Medicine

Research output: Contribution to journal › Short survey › peer-review

10 Scopus citations

Abstract

Tumors cells can release natural killer (NK) cell ligands for activating receptor NKG2D that are thought to inhibit NK cell function. In a recent issue of Science, Deng etal. (2015) show that, unexpectedly, a soluble NKG2D ligand can enhance anti-tumor NK cell activity.

Original language	English (US)
Pages (from-to)	605-606
Number of pages	2
Journal	Immunity
Volume	42
Issue number	4
DOIs	https://doi.org/10.1016/j.immuni.2015.04.007
State	Published - Apr 21 2015

ASJC Scopus subject areas

Immunology and Allergy
Immunology
Infectious Diseases

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10.1016/j.immuni.2015.04.007

Cite this

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title = "Distinct Mechanisms of Tumor Resistance to NK Killing: Of Mice and Men",

abstract = "Tumors cells can release natural killer (NK) cell ligands for activating receptor NKG2D that are thought to inhibit NK cell function. In a recent issue of Science, Deng etal. (2015) show that, unexpectedly, a soluble NKG2D ligand can enhance anti-tumor NK cell activity.",

author = "Pardoll, {Drew M.}",

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year = "2015",

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AU - Pardoll, Drew M.

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AB - Tumors cells can release natural killer (NK) cell ligands for activating receptor NKG2D that are thought to inhibit NK cell function. In a recent issue of Science, Deng etal. (2015) show that, unexpectedly, a soluble NKG2D ligand can enhance anti-tumor NK cell activity.

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M3 - Short survey

C2 - 25902479

AN - SCOPUS:84928202804

SN - 1074-7613

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SP - 605

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JO - Immunity

JF - Immunity

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ER -

Distinct Mechanisms of Tumor Resistance to NK Killing: Of Mice and Men

Abstract

ASJC Scopus subject areas

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