TY - JOUR
T1 - Corrigendum
T2 - Editorial: Cross-talk between heterogeneous cell types in skeletal muscle: implications for glucose metabolism(Front. Endocrinol., (2023), 14, (1185725), 10.3389/fendo.2023.1185725)
AU - Caballero-Sánchez, Noemí
AU - Winn, Nathan
AU - Rosa Neto, Jose Cesar
AU - Nagy, Laszlo
N1 - Publisher Copyright:
Copyright © 2023 Caballero-Sánchez, Winn, Rosa Neto and Nagy.
PY - 2023
Y1 - 2023
N2 - In the published article, Figure 1 and the respective citations were omitted. The corrected Figure 1 and its caption appear below. Glucose metabolism associated pathways and its correlation with growth, inflammation, and tissue repair in skeletal muscle. The citations for Figure 1 have now been inserted in paragraphs 3 and 4 and should read: “In a recently published paper, Merz et al. link the relevance of PAK1 to type II diabetes pathogenesis by the use of knockout and overexpression of PAK1 mouse models (Figure 1).” “Nonetheless, the interconnection between insulin release, resistance, and glucose metabolism in skeletal muscle is much more complex and it also has an immunological cofactor as reviewed by Shen et al. linking the systematic pro-inflammatory response detected by several studies by the increased levels of TNFa or IL-6 in serum (8) and intramuscular (9) with muscle degeneration (Figure 1).” “In this issue, Arias-Calderoín et al. have been able to identify that FGF21 an important hormone for muscle repair, can be secreted intra-muscularly via PI3K/AKT/mTOR signaling pathway (Figure 1).” The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.
AB - In the published article, Figure 1 and the respective citations were omitted. The corrected Figure 1 and its caption appear below. Glucose metabolism associated pathways and its correlation with growth, inflammation, and tissue repair in skeletal muscle. The citations for Figure 1 have now been inserted in paragraphs 3 and 4 and should read: “In a recently published paper, Merz et al. link the relevance of PAK1 to type II diabetes pathogenesis by the use of knockout and overexpression of PAK1 mouse models (Figure 1).” “Nonetheless, the interconnection between insulin release, resistance, and glucose metabolism in skeletal muscle is much more complex and it also has an immunological cofactor as reviewed by Shen et al. linking the systematic pro-inflammatory response detected by several studies by the increased levels of TNFa or IL-6 in serum (8) and intramuscular (9) with muscle degeneration (Figure 1).” “In this issue, Arias-Calderoín et al. have been able to identify that FGF21 an important hormone for muscle repair, can be secreted intra-muscularly via PI3K/AKT/mTOR signaling pathway (Figure 1).” The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.
KW - FGF21
KW - glucose metabolism
KW - inflammation
KW - skeletal muscle
KW - tissue cross talk
UR - http://www.scopus.com/inward/record.url?scp=85164701055&partnerID=8YFLogxK
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U2 - 10.3389/fendo.2023.1219036
DO - 10.3389/fendo.2023.1219036
M3 - Comment/debate
C2 - 37435489
AN - SCOPUS:85164701055
SN - 1664-2392
VL - 14
JO - Frontiers in Endocrinology
JF - Frontiers in Endocrinology
M1 - 1219036
ER -