Author Correction: Defects in mtDNA replication challenge nuclear genome stability through nucleotide depletion and provide a unifying mechanism for mouse progerias (Nature Metabolism, (2019), 1, 10, (958-965), 10.1038/s42255-019-0120-1)

Riikka H. Hämäläinen; Juan C. Landoni; Kati J. Ahlqvist; Steffi Goffart; Sanna Ryytty; M. Obaidur Rahman; Virginia Brilhante; Katherine Icay; Sampsa Hautaniemi; Liya Wang; Marikki Laiho; Anu Suomalainen

doi:10.1038/s42255-020-0255-0

Author Correction: Defects in mtDNA replication challenge nuclear genome stability through nucleotide depletion and provide a unifying mechanism for mouse progerias (Nature Metabolism, (2019), 1, 10, (958-965), 10.1038/s42255-019-0120-1)

Riikka H. Hämäläinen
, Juan C. Landoni
, Kati J. Ahlqvist
, Steffi Goffart
, Sanna Ryytty
, M. Obaidur Rahman
, Virginia Brilhante
, Katherine Icay
, Sampsa Hautaniemi
, Liya Wang
, Marikki Laiho
, Anu Suomalainen

School of Medicine

Research output: Contribution to journal › Comment/debate › peer-review

Abstract

In the version of this article initially published, in Fig. 2c,d and Extended Data Fig. 3a,b, the y axes of the dNTP plots were mislabelled. In Fig. 2c,d, the y-axis labels read ‘[dNTP] relative’ but should have read ‘[dNTP] fmol per μg mitochondrial protein’. In Extended Data Fig. 3a,b, the y-axis units read ‘[dNTP] uM’ but should have read ‘[dNTP] nmol/million cells’. The errors have been corrected in the HTML and PDF versions of the article.

Original language	English (US)
Pages (from-to)	793
Number of pages	1
Journal	Nature Metabolism
Volume	2
Issue number	8
DOIs	https://doi.org/10.1038/s42255-020-0255-0
State	Published - Aug 1 2020

ASJC Scopus subject areas

Endocrinology, Diabetes and Metabolism
Physiology (medical)
Internal Medicine
Cell Biology
General Medicine

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Hämäläinen, R. H., Landoni, J. C., Ahlqvist, K. J., Goffart, S., Ryytty, S., Rahman, M. O., Brilhante, V., Icay, K., Hautaniemi, S., Wang, L., Laiho, M., & Suomalainen, A. (2020). Author Correction: Defects in mtDNA replication challenge nuclear genome stability through nucleotide depletion and provide a unifying mechanism for mouse progerias (Nature Metabolism, (2019), 1, 10, (958-965), 10.1038/s42255-019-0120-1). Nature Metabolism, 2(8), 793. https://doi.org/10.1038/s42255-020-0255-0

Author Correction: Defects in mtDNA replication challenge nuclear genome stability through nucleotide depletion and provide a unifying mechanism for mouse progerias (Nature Metabolism, (2019), 1, 10, (958-965), 10.1038/s42255-019-0120-1). / Hämäläinen, Riikka H.; Landoni, Juan C.; Ahlqvist, Kati J. et al.
In: Nature Metabolism, Vol. 2, No. 8, 01.08.2020, p. 793.

Research output: Contribution to journal › Comment/debate › peer-review

Hämäläinen, RH, Landoni, JC, Ahlqvist, KJ, Goffart, S, Ryytty, S, Rahman, MO, Brilhante, V, Icay, K, Hautaniemi, S, Wang, L, Laiho, M & Suomalainen, A 2020, 'Author Correction: Defects in mtDNA replication challenge nuclear genome stability through nucleotide depletion and provide a unifying mechanism for mouse progerias (Nature Metabolism, (2019), 1, 10, (958-965), 10.1038/s42255-019-0120-1)', Nature Metabolism, vol. 2, no. 8, pp. 793. https://doi.org/10.1038/s42255-020-0255-0

Hämäläinen RH, Landoni JC, Ahlqvist KJ, Goffart S, Ryytty S, Rahman MO et al. Author Correction: Defects in mtDNA replication challenge nuclear genome stability through nucleotide depletion and provide a unifying mechanism for mouse progerias (Nature Metabolism, (2019), 1, 10, (958-965), 10.1038/s42255-019-0120-1). Nature Metabolism. 2020 Aug 1;2(8):793. doi: 10.1038/s42255-020-0255-0

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title = "Author Correction: Defects in mtDNA replication challenge nuclear genome stability through nucleotide depletion and provide a unifying mechanism for mouse progerias (Nature Metabolism, (2019), 1, 10, (958-965), 10.1038/s42255-019-0120-1)",

abstract = "In the version of this article initially published, in Fig. 2c,d and Extended Data Fig. 3a,b, the y axes of the dNTP plots were mislabelled. In Fig. 2c,d, the y-axis labels read {\textquoteleft}[dNTP] relative{\textquoteright} but should have read {\textquoteleft}[dNTP] fmol per μg mitochondrial protein{\textquoteright}. In Extended Data Fig. 3a,b, the y-axis units read {\textquoteleft}[dNTP] uM{\textquoteright} but should have read {\textquoteleft}[dNTP] nmol/million cells{\textquoteright}. The errors have been corrected in the HTML and PDF versions of the article.",

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AU - Hämäläinen, Riikka H.

AU - Landoni, Juan C.

AU - Ahlqvist, Kati J.

AU - Goffart, Steffi

AU - Ryytty, Sanna

AU - Rahman, M. Obaidur

AU - Brilhante, Virginia

AU - Icay, Katherine

AU - Hautaniemi, Sampsa

AU - Wang, Liya

AU - Laiho, Marikki

AU - Suomalainen, Anu

PY - 2020/8/1

Y1 - 2020/8/1

N2 - In the version of this article initially published, in Fig. 2c,d and Extended Data Fig. 3a,b, the y axes of the dNTP plots were mislabelled. In Fig. 2c,d, the y-axis labels read ‘[dNTP] relative’ but should have read ‘[dNTP] fmol per μg mitochondrial protein’. In Extended Data Fig. 3a,b, the y-axis units read ‘[dNTP] uM’ but should have read ‘[dNTP] nmol/million cells’. The errors have been corrected in the HTML and PDF versions of the article.

AB - In the version of this article initially published, in Fig. 2c,d and Extended Data Fig. 3a,b, the y axes of the dNTP plots were mislabelled. In Fig. 2c,d, the y-axis labels read ‘[dNTP] relative’ but should have read ‘[dNTP] fmol per μg mitochondrial protein’. In Extended Data Fig. 3a,b, the y-axis units read ‘[dNTP] uM’ but should have read ‘[dNTP] nmol/million cells’. The errors have been corrected in the HTML and PDF versions of the article.

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SN - 2522-5812

VL - 2

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JO - Nature Metabolism

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Author Correction: Defects in mtDNA replication challenge nuclear genome stability through nucleotide depletion and provide a unifying mechanism for mouse progerias (Nature Metabolism, (2019), 1, 10, (958-965), 10.1038/s42255-019-0120-1)

Abstract

ASJC Scopus subject areas

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