Author Correction: Defects in mtDNA replication challenge nuclear genome stability through nucleotide depletion and provide a unifying mechanism for mouse progerias (Nature Metabolism, (2019), 1, 10, (958-965), 10.1038/s42255-019-0120-1)

Riikka H. Hämäläinen; Juan C. Landoni; Kati J. Ahlqvist; Steffi Goffart; Sanna Ryytty; M. Obaidur Rahman; Virginia Brilhante; Katherine Icay; Sampsa Hautaniemi; Liya Wang; Marikki Laiho; Anu Suomalainen

doi:10.1038/s42255-020-0255-0

Author Correction: Defects in mtDNA replication challenge nuclear genome stability through nucleotide depletion and provide a unifying mechanism for mouse progerias (Nature Metabolism, (2019), 1, 10, (958-965), 10.1038/s42255-019-0120-1)

Riikka H. Hämäläinen, Juan C. Landoni, Kati J. Ahlqvist, Steffi Goffart, Sanna Ryytty, M. Obaidur Rahman, Virginia Brilhante, Katherine Icay, Sampsa Hautaniemi, Liya Wang, Marikki Laiho, Anu Suomalainen

School of Medicine

Research output: Contribution to journal › Comment/debate › peer-review

Abstract

In the version of this article initially published, in Fig. 2c,d and Extended Data Fig. 3a,b, the y axes of the dNTP plots were mislabelled. In Fig. 2c,d, the y-axis labels read ‘[dNTP] relative’ but should have read ‘[dNTP] fmol per μg mitochondrial protein’. In Extended Data Fig. 3a,b, the y-axis units read ‘[dNTP] uM’ but should have read ‘[dNTP] nmol/million cells’. The errors have been corrected in the HTML and PDF versions of the article.

Original language	English (US)
Pages (from-to)	793
Number of pages	1
Journal	Nature Metabolism
Volume	2
Issue number	8
DOIs	https://doi.org/10.1038/s42255-020-0255-0
State	Published - Aug 1 2020

ASJC Scopus subject areas

Endocrinology, Diabetes and Metabolism
Physiology (medical)
Internal Medicine
Cell Biology
Medicine(all)

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Hämäläinen, R. H., Landoni, J. C., Ahlqvist, K. J., Goffart, S., Ryytty, S., Rahman, M. O., Brilhante, V., Icay, K., Hautaniemi, S., Wang, L., Laiho, M., & Suomalainen, A. (2020). Author Correction: Defects in mtDNA replication challenge nuclear genome stability through nucleotide depletion and provide a unifying mechanism for mouse progerias (Nature Metabolism, (2019), 1, 10, (958-965), 10.1038/s42255-019-0120-1). Nature Metabolism, 2(8), 793. https://doi.org/10.1038/s42255-020-0255-0

Author Correction: Defects in mtDNA replication challenge nuclear genome stability through nucleotide depletion and provide a unifying mechanism for mouse progerias (Nature Metabolism, (2019), 1, 10, (958-965), 10.1038/s42255-019-0120-1). / Hämäläinen, Riikka H.; Landoni, Juan C.; Ahlqvist, Kati J. et al.
In: Nature Metabolism, Vol. 2, No. 8, 01.08.2020, p. 793.

Research output: Contribution to journal › Comment/debate › peer-review

Hämäläinen, RH, Landoni, JC, Ahlqvist, KJ, Goffart, S, Ryytty, S, Rahman, MO, Brilhante, V, Icay, K, Hautaniemi, S, Wang, L, Laiho, M & Suomalainen, A 2020, 'Author Correction: Defects in mtDNA replication challenge nuclear genome stability through nucleotide depletion and provide a unifying mechanism for mouse progerias (Nature Metabolism, (2019), 1, 10, (958-965), 10.1038/s42255-019-0120-1)', Nature Metabolism, vol. 2, no. 8, pp. 793. https://doi.org/10.1038/s42255-020-0255-0

Hämäläinen RH, Landoni JC, Ahlqvist KJ, Goffart S, Ryytty S, Rahman MO et al. Author Correction: Defects in mtDNA replication challenge nuclear genome stability through nucleotide depletion and provide a unifying mechanism for mouse progerias (Nature Metabolism, (2019), 1, 10, (958-965), 10.1038/s42255-019-0120-1). Nature Metabolism. 2020 Aug 1;2(8):793. doi: 10.1038/s42255-020-0255-0

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abstract = "In the version of this article initially published, in Fig. 2c,d and Extended Data Fig. 3a,b, the y axes of the dNTP plots were mislabelled. In Fig. 2c,d, the y-axis labels read {\textquoteleft}[dNTP] relative{\textquoteright} but should have read {\textquoteleft}[dNTP] fmol per μg mitochondrial protein{\textquoteright}. In Extended Data Fig. 3a,b, the y-axis units read {\textquoteleft}[dNTP] uM{\textquoteright} but should have read {\textquoteleft}[dNTP] nmol/million cells{\textquoteright}. The errors have been corrected in the HTML and PDF versions of the article.",

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AU - Hämäläinen, Riikka H.

AU - Landoni, Juan C.

AU - Ahlqvist, Kati J.

AU - Goffart, Steffi

AU - Ryytty, Sanna

AU - Rahman, M. Obaidur

AU - Brilhante, Virginia

AU - Icay, Katherine

AU - Hautaniemi, Sampsa

AU - Wang, Liya

AU - Laiho, Marikki

AU - Suomalainen, Anu

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N2 - In the version of this article initially published, in Fig. 2c,d and Extended Data Fig. 3a,b, the y axes of the dNTP plots were mislabelled. In Fig. 2c,d, the y-axis labels read ‘[dNTP] relative’ but should have read ‘[dNTP] fmol per μg mitochondrial protein’. In Extended Data Fig. 3a,b, the y-axis units read ‘[dNTP] uM’ but should have read ‘[dNTP] nmol/million cells’. The errors have been corrected in the HTML and PDF versions of the article.

AB - In the version of this article initially published, in Fig. 2c,d and Extended Data Fig. 3a,b, the y axes of the dNTP plots were mislabelled. In Fig. 2c,d, the y-axis labels read ‘[dNTP] relative’ but should have read ‘[dNTP] fmol per μg mitochondrial protein’. In Extended Data Fig. 3a,b, the y-axis units read ‘[dNTP] uM’ but should have read ‘[dNTP] nmol/million cells’. The errors have been corrected in the HTML and PDF versions of the article.

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Author Correction: Defects in mtDNA replication challenge nuclear genome stability through nucleotide depletion and provide a unifying mechanism for mouse progerias (Nature Metabolism, (2019), 1, 10, (958-965), 10.1038/s42255-019-0120-1)

Abstract

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