Adolescent stress leads to glutamatergic disturbance through dopaminergic abnormalities in the prefrontal cortex of genetically vulnerable mice

Keyphrases

• Adolescent Period 20%
• Adolescent Stress 100%
• Adult Stage 20%
• Adulthood 20%
• Atypical Antipsychotics 20%
• Background Stress 20%
• Behavioral Deficits 40%
• Behavioral Patterns 20%
• Ca2+ 20%
• CaMKII 20%
• Clinical Features 20%
• Clozapine 20%
• DISC1 20%
• Dopamine D1 Agonists 20%
• Dopaminergic Neurons 20%
• Forced Swim 20%
• Functional Change 20%
• Genetic Risk 20%
• Glutamate 20%
• Glutamate Transporter 1 (GLT-1) 20%
• Glutamatergic 100%
• Glutamatergic Neurons 20%
• Glutamatergic System 40%
• Interaction Preference 20%
• Isolation Stress 20%
• Late Adolescents 20%
• Locomotor Activity 20%
• Molecular Changes 40%
• N-methyl-D-aspartate 20%
• N-methyl-D-aspartate Receptor (NMDAR) 20%
• Novelty Preference 20%
• Older Adults 20%
• Postnatal Maturation 20%
• Preference Test 20%
• Prefrontal Cortex 100%
• Psychiatric Disorders 20%
• Schizophrenia 20%
• SKF81297 20%
• Social Interaction 20%
• Social Novelty 20%
• Stress-induced 20%

Neuroscience

• Agonist 20%
• Atypical Antipsychotic 20%
• Calcium Calmodulin Dependent Protein Kinase II 20%
• Clozapine 20%
• Dementia Praecox 40%
• Dextro Aspartic Acid 40%
• Dextro Serine 20%
• Dopaminergic 100%
• Dopaminergic Neuron 20%
• Glutamate Transporter 20%
• Glutamic Acid 20%
• Isolation Stress 20%
• Locomotor Activity 20%
• Methyldienolone 40%
• Prefrontal Cortex 100%
• Social Interaction 20%
• Typical Antipsychotic 20%