A new trick for cyclin-cdk: Activation of STAT

Debra L. Silver, Denise J. Montell

Research output: Contribution to journalReview articlepeer-review

6 Scopus citations


New work in Drosophila demonstrates that cdk4 loss causes phenotypes similar to the loss of JAK/STAT pathway components. Cdk4 overexpression can bypass requirements for JAK but not STAT. These results demonstrate a new function for Cdk4 and a new mode of STAT activation.

Original languageEnglish (US)
Pages (from-to)148-149
Number of pages2
JournalDevelopmental Cell
Issue number2
StatePublished - Feb 1 2003
Externally publishedYes

ASJC Scopus subject areas

  • Molecular Biology
  • Biochemistry, Genetics and Molecular Biology(all)
  • Developmental Biology
  • Cell Biology


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