A Bcl-xL Timer Sets Platelet Life Span

Research output: Contribution to journalShort surveypeer-review

21 Scopus citations


Platelets are cell fragments lacking nuclei that play a key role in blood clotting. Using an impressive genetic screen involving ENU-mutagenesis of whole mice, Mason et al. (2007) report in this issue their identification of mutations in the antiapoptotic protein Bcl-xL that cause accelerated death of platelets leading to platelet deficiency.

Original languageEnglish (US)
Pages (from-to)1035-1036
Number of pages2
Issue number6
StatePublished - Mar 23 2007

ASJC Scopus subject areas

  • General Biochemistry, Genetics and Molecular Biology


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